Blockade of NMDA receptors and apoptotic neurodegeneration in the developing brain. Science 1999 Jan 1;283(5398):70-4. Ikonomidou C, Bosch F, Miksa M, Bittigau P, Vockler J, Dikranian K, Tenkova TI, Stefovska V, Turski L, Olney JW. C. Ikonomidou, F. Bosch, M. Miksa, P. Bittigau, J. Vockler, V. Stefovska, Department of Pediatric Neurology, Charite-Virchow Clinics, Humboldt University, Augustenburger Platz 1, 13353 Berlin, Germany. K. Dikranian, T. I. Tenkova, J. W. Ol. ["Blockade of NMDA receptors gives rise to different patterns of neuronal loss depending on the stage of development at which the interference occurs. Such a mechanism could contribute to a variety of neuropsychiatric disorders."]
Role of heteromer formation in GABAB receptor function. Science 1999 Jan 1;283(5398):74-7. Kuner R, Kohr G, Gr newald S, Eisenhardt G, Bach A, Kornau HC. R. Kuner, S. Grunewald, G. Eisenhardt, A. Bach, H.-C. Kornau, BASF-LYNX Bioscience AG, Department of Neuroscience, Im Neuenheimer Feld 515, D-69120 Heidelberg, Germany. G. Kohr, Max-Planck-Institute for Medical Research, Dep.
Non-cholinergic, trophic action of recombinant acetylcholinesterase on mid-brain dopaminergic neurons. J Neurosci Res 1997 Jul 15;49(2):207-18.Holmes C, Jones SA, Budd TC, Greenfield SA. University Department of Pharmacology, Oxford University, United Kingdom.